
Ping. You check your phone and see a notification from your friend, unlocking it to see what they have sent. Before you know it, 30 minutes have passed, and there is now a cat spinning at insane speed with high-pitched background music on your phone – how did you get here? You have forgotten what you wanted to do in the first place, and if this sounds familiar, you could be wondering why it’s getting seemingly more difficult to focus these days. Was Mom right – could it really be the phone’s fault? Or have we gotten ‘lazier’ as a generation?
Ping. You check your phone and see a notification from your friend, unlocking it to see what they have sent. Before you know it, 30 minutes have passed, and there is now a cat spinning at insane speed with high-pitched background music on your phone – how did you get here? You have forgotten what you wanted to do in the first place, and if this sounds familiar, you could be wondering why it’s getting seemingly more difficult to focus these days. Was Mom right – could it really be the phone’s fault? Or have we gotten ‘lazier’ as a generation?
Photo by Russlan Kossatschjow

Photo by Russlan Kossatschjow
To begin answering these questions, it is crucial to learn about the role of dopamine, which is the key neurotransmitter in the regulation of our motivation, focus, and reinforcement learning (Olguín et al., 2015). More importantly, dopamine plays a significant part in processing present rewards and predicting future ones, thus contributing to our capacity for delayed gratification (Schultz, 2016). Quick-hit moments like drinking alcohol, online shopping, cannabis usage (Uhl et al., 2019), and the (in)famously dreaded doom scrolling, all can seem harmless, but in reality, they only provide temporary surges of dopamine that typically wear off within a few hours. Such activities can leave us craving more and more of the “pleasurable” experience while simultaneously making it harder to concentrate on tasks that offer less immediate gratification, yet are important in our day‑to‑day lives. It’s important to note that this is not exactly a failure of willpower but rather, behavioural changes stemming from a biological adaptation to an environment that is oversaturated with distractions.
To see how these behavioral changes emerge from our biology, we need to zoom in on the specific brain regions where dopamine does its work. The central areas upon where the chemical acts are the Ventral Tegmental Area (VTA), insular cortex, and nucleus accumbens (NAcc), which are all part of the reward pathway (Li & Jasanoff, 2020). The mesolimbic dopamine pathway, which originates in the VTA, is mainly associated with evaluating the salience and novelty of stimuli (Stott & Ang, 2013) and then it goes on to predict rewards and adjusts our behaviour accordingly. This reward prediction process is central to what is known as the dopamine feedback loop – when we encounter a stimulus which we expect to bring rewards, dopaminergic levels will rise in anticipation (Stolyarova et al., 2014). If the outcome of the reward matches what we expected, then this causes the feedback loop to be strengthened and, in turn, the behaviour itself. The opposite is true if the expectation of the reward does not match its actual outcome, in turn weakening the feedback loop. Moreover, an imbalance of dopamine signals in the prefrontal cortex, which is the region responsible for planning, impulse control, and working memory, further compromises our ability to focus (Ott & Nieder, 2019). What feels like laziness or procrastination is often the result of a brain that is conditioned to operate under a different set of expectations, one that constantly scans for novel stimuli and resists cognitive effort – turning into a cognitive ‘miser’ instead. Rather than sustaining attempts toward long-term rewards (e.g., finishing a book, developing a hobby, or perfecting a skill), the system becomes used to seeking immediate and low-effort surges of gratification. This repeated overstimulation begins to desensitize dopamine receptors and lowers the threshold for what the brain considers engaging or worth pursuing (Burhan & Moradzadeh, 2020).
“What feels like laziness or procrastination is often the result of a brain that is conditioned to operate under a different set of expectations, one that constantly scans for novel stimuli and resists cognitive effort - turning into a cognitive ‘miser’ instead.”
Chronic overstimulation of the brain’s reward system does more than just affect focus. Research by Dalley & Roiser (2012) links dopamine dysregulation to impulsivity, not just in decision-making, but also in mood instability, task-switching, and emotional reactivity. This physically manifests in the tendency to prematurely abandon tasks and decreased frustration tolerance, i.e. the degree to which one is able to handle frustrating situations. The brain increasingly prioritizes the dopaminergic salience of activities rather than their intrinsic meaning. Such shifts in the balance of dopamine are also reflected in our social lives, where the drive for rapid gratification can make us much more emotionally reactive (Seo et al., 2008). Surprisingly, too much or too little of this neurotransmitter can even determine how willing we are to be invested in building relationships, which require continuous effort in the long run; even conversations can start to feel draining if they lack novelty (Raji et al., 2025).
“Fortunately, while it’s true that the dopamine system is pliant, it does not mean its dysregulation is fixed. One promising approach for reversing the overload of this chemical is ‘dopamine fasting’. ”
Fortunately, while it’s true that the dopamine system is pliant, it does not mean its dysregulation is fixed. One promising approach for reversing the overload of this chemical is ‘dopamine fasting’. Though the name can seem misleading, it does not involve starving the brain of dopamine itself, but rather, it refers to reducing the exposure to the digitally saturated world – this allows the reward pathways time away from high frequencies and to recalibrate itself (Desai et al., 2024). The goal is to help the brain tune itself to slower and more natural sources of reward by carving out time without social media, streaming services, online purchases, etc. Recalibration also depends on gradually reintroducing activities that engage the reward system in a more sustainable way while simultaneously reinforcing them. For example, focused reading or physical exercise can challenge the brain’s attention mechanisms, encouraging it to rebuild pathways associated with sustained concentration and delayed gratification (Bastioli et al., 2022). In addition, time spent in natural environments has been shown to reduce cortisol and promote recovery in the prefrontal cortex, offering a calm source of positive feedback in the dopamine loop (Darna et al., 2014).
Nonetheless, rewiring a dopamine system conditioned for rapid spikes takes time. Early stages of withdrawal from overstimulation can often feel uncomfortable. However,with consistency, the neural circuitry begins to shift. Tasks that once felt effortful or boring regain their appeal, attention span lengthens, and the mind becomes more capable of engaging deeply without needing constant novelty to stay motivated. Your brain is not broken for chasing every notification, especially in a world that is wired for speed and stimulation – perhaps a quiet walk or a book chapter may be a new form of rebellion.
References
- Bastioli, G., Arnold, J. C., Mancini, M., Mar, A. C., Gamallo-Lana, B., Saadipour, K., Chao, M. V., & Rice, M. E. (2022). Voluntary exercise boosts striatal dopamine release: evidence for the necessary and sufficient role of BDNF. Journal of Neuroscience, 42(23), 4725–4736. https://doi.org/10.1523/jneurosci.2273-21.2022
- Burhan, R., & Moradzadeh, J. (2020). Neurotransmitter Dopamine (DA) and its Role in the Development of Social Media Addiction. Journal of Neurology & Neurophysiology, 11(7), 1–2. https://doi.org/10.35248/2155-9562.20.11.507
- Dalley, J., & Roiser, J. (2012). Dopamine, serotonin and impulsivity. Neuroscience, 215, 42–58. https://doi.org/10.1016/j.neuroscience.2012.03.065
- Darna, M., Beckmann, J. S., Gipson, C. D., Bardo, M. T., & Dwoskin, L. P. (2014). Effect of environmental enrichment on dopamine and serotonin transporters and glutamate neurotransmission in medial prefrontal and orbitofrontal cortex. Brain Research, 1599, 115–125. https://doi.org/10.1016/j.brainres.2014.12.034
- Desai, D., Patel, J., Saiyed, F., Upadhyay, H., Kariya, P., & Patel, J. (2024). A Literature Review on Holistic Well-Being and Dopamine Fasting: An Integrated Approach. Cureus. https://doi.org/10.7759/cureus.61643
- Li, N., & Jasanoff, A. (2020). Local and global consequences of reward-evoked striatal dopamine release. Nature, 580(7802), 239–244. https://doi.org/10.1038/s41586-020-2158-3
- Olguín, H. J., Guzmán, D. C., García, E. H., & Mejía, G. B. (2015). The role of dopamine and its dysfunction as a consequence of oxidative stress. Oxidative Medicine and Cellular Longevity, 2016(1). https://doi.org/10.1155/2016/9730467
- Ott, T., & Nieder, A. (2019). Dopamine and cognitive control in prefrontal cortex. Trends in Cognitive Sciences, 23(3), 213–234. https://doi.org/10.1016/j.tics.2018.12.006
- Raji, H., Dinesh, S., & Sharma, S. (2025). Inside the impulsive brain: a narrative review on the role of neurobiological, hormonal and genetic factors influencing impulsivity in psychiatric disorders. The Egyptian Journal of Neurology Psychiatry and Neurosurgery, 61(1). https://doi.org/10.1186/s41983-024-00930-9
- Schultz, W. (2016). Dopamine reward prediction error coding. Dialogues in Clinical Neuroscience, 18(1), 23–32. https://doi.org/10.31887/dcns.2016.18.1/wschultz
- Seo, D., Patrick, C. J., & Kennealy, P. J. (2008). Role of serotonin and dopamine system interactions in the neurobiology of impulsive aggression and its comorbidity with other clinical disorders. Aggression and Violent Behavior, 13(5), 383–395. https://doi.org/10.1016/j.avb.2008.06.003
- Stolyarova, A., O’Dell, S. J., Marshall, J. F., & Izquierdo, A. (2014). Positive and negative feedback learning and associated dopamine and serotonin transporter binding after methamphetamine. Behavioural Brain Research, 271, 195–202. https://doi.org/10.1016/j.bbr.2014.06.031
- Stott, S., & Ang, S. (2013). The generation of midbrain dopaminergic neurons. In Elsevier eBooks (pp. 435–453). https://doi.org/10.1016/b978-0-12-397265-1.00099-x
- Uhl, G. R., Koob, G. F., & Cable, J. (2019). The neurobiology of addiction. Annals of the New York Academy of Sciences, 1451(1), 5–28. https://doi.org/10.1111/nyas.13989
To begin answering these questions, it is crucial to learn about the role of dopamine, which is the key neurotransmitter in the regulation of our motivation, focus, and reinforcement learning (Olguín et al., 2015). More importantly, dopamine plays a significant part in processing present rewards and predicting future ones, thus contributing to our capacity for delayed gratification (Schultz, 2016). Quick-hit moments like drinking alcohol, online shopping, cannabis usage (Uhl et al., 2019), and the (in)famously dreaded doom scrolling, all can seem harmless, but in reality, they only provide temporary surges of dopamine that typically wear off within a few hours. Such activities can leave us craving more and more of the “pleasurable” experience while simultaneously making it harder to concentrate on tasks that offer less immediate gratification, yet are important in our day‑to‑day lives. It’s important to note that this is not exactly a failure of willpower but rather, behavioural changes stemming from a biological adaptation to an environment that is oversaturated with distractions.
To see how these behavioral changes emerge from our biology, we need to zoom in on the specific brain regions where dopamine does its work. The central areas upon where the chemical acts are the Ventral Tegmental Area (VTA), insular cortex, and nucleus accumbens (NAcc), which are all part of the reward pathway (Li & Jasanoff, 2020). The mesolimbic dopamine pathway, which originates in the VTA, is mainly associated with evaluating the salience and novelty of stimuli (Stott & Ang, 2013) and then it goes on to predict rewards and adjusts our behaviour accordingly. This reward prediction process is central to what is known as the dopamine feedback loop – when we encounter a stimulus which we expect to bring rewards, dopaminergic levels will rise in anticipation (Stolyarova et al., 2014). If the outcome of the reward matches what we expected, then this causes the feedback loop to be strengthened and, in turn, the behaviour itself. The opposite is true if the expectation of the reward does not match its actual outcome, in turn weakening the feedback loop. Moreover, an imbalance of dopamine signals in the prefrontal cortex, which is the region responsible for planning, impulse control, and working memory, further compromises our ability to focus (Ott & Nieder, 2019). What feels like laziness or procrastination is often the result of a brain that is conditioned to operate under a different set of expectations, one that constantly scans for novel stimuli and resists cognitive effort – turning into a cognitive ‘miser’ instead. Rather than sustaining attempts toward long-term rewards (e.g., finishing a book, developing a hobby, or perfecting a skill), the system becomes used to seeking immediate and low-effort surges of gratification. This repeated overstimulation begins to desensitize dopamine receptors and lowers the threshold for what the brain considers engaging or worth pursuing (Burhan & Moradzadeh, 2020).
“What feels like laziness or procrastination is often the result of a brain that is conditioned to operate under a different set of expectations, one that constantly scans for novel stimuli and resists cognitive effort - turning into a cognitive ‘miser’ instead.”
Chronic overstimulation of the brain’s reward system does more than just affect focus. Research by Dalley & Roiser (2012) links dopamine dysregulation to impulsivity, not just in decision-making, but also in mood instability, task-switching, and emotional reactivity. This physically manifests in the tendency to prematurely abandon tasks and decreased frustration tolerance, i.e. the degree to which one is able to handle frustrating situations. The brain increasingly prioritizes the dopaminergic salience of activities rather than their intrinsic meaning. Such shifts in the balance of dopamine are also reflected in our social lives, where the drive for rapid gratification can make us much more emotionally reactive (Seo et al., 2008). Surprisingly, too much or too little of this neurotransmitter can even determine how willing we are to be invested in building relationships, which require continuous effort in the long run; even conversations can start to feel draining if they lack novelty (Raji et al., 2025).
“Fortunately, while it’s true that the dopamine system is pliant, it does not mean its dysregulation is fixed. One promising approach for reversing the overload of this chemical is ‘dopamine fasting’. ”
Fortunately, while it’s true that the dopamine system is pliant, it does not mean its dysregulation is fixed. One promising approach for reversing the overload of this chemical is ‘dopamine fasting’. Though the name can seem misleading, it does not involve starving the brain of dopamine itself, but rather, it refers to reducing the exposure to the digitally saturated world – this allows the reward pathways time away from high frequencies and to recalibrate itself (Desai et al., 2024). The goal is to help the brain tune itself to slower and more natural sources of reward by carving out time without social media, streaming services, online purchases, etc. Recalibration also depends on gradually reintroducing activities that engage the reward system in a more sustainable way while simultaneously reinforcing them. For example, focused reading or physical exercise can challenge the brain’s attention mechanisms, encouraging it to rebuild pathways associated with sustained concentration and delayed gratification (Bastioli et al., 2022). In addition, time spent in natural environments has been shown to reduce cortisol and promote recovery in the prefrontal cortex, offering a calm source of positive feedback in the dopamine loop (Darna et al., 2014).
Nonetheless, rewiring a dopamine system conditioned for rapid spikes takes time. Early stages of withdrawal from overstimulation can often feel uncomfortable. However,with consistency, the neural circuitry begins to shift. Tasks that once felt effortful or boring regain their appeal, attention span lengthens, and the mind becomes more capable of engaging deeply without needing constant novelty to stay motivated. Your brain is not broken for chasing every notification, especially in a world that is wired for speed and stimulation – perhaps a quiet walk or a book chapter may be a new form of rebellion.
References
- Bastioli, G., Arnold, J. C., Mancini, M., Mar, A. C., Gamallo-Lana, B., Saadipour, K., Chao, M. V., & Rice, M. E. (2022). Voluntary exercise boosts striatal dopamine release: evidence for the necessary and sufficient role of BDNF. Journal of Neuroscience, 42(23), 4725–4736. https://doi.org/10.1523/jneurosci.2273-21.2022
- Burhan, R., & Moradzadeh, J. (2020). Neurotransmitter Dopamine (DA) and its Role in the Development of Social Media Addiction. Journal of Neurology & Neurophysiology, 11(7), 1–2. https://doi.org/10.35248/2155-9562.20.11.507
- Dalley, J., & Roiser, J. (2012). Dopamine, serotonin and impulsivity. Neuroscience, 215, 42–58. https://doi.org/10.1016/j.neuroscience.2012.03.065
- Darna, M., Beckmann, J. S., Gipson, C. D., Bardo, M. T., & Dwoskin, L. P. (2014). Effect of environmental enrichment on dopamine and serotonin transporters and glutamate neurotransmission in medial prefrontal and orbitofrontal cortex. Brain Research, 1599, 115–125. https://doi.org/10.1016/j.brainres.2014.12.034
- Desai, D., Patel, J., Saiyed, F., Upadhyay, H., Kariya, P., & Patel, J. (2024). A Literature Review on Holistic Well-Being and Dopamine Fasting: An Integrated Approach. Cureus. https://doi.org/10.7759/cureus.61643
- Li, N., & Jasanoff, A. (2020). Local and global consequences of reward-evoked striatal dopamine release. Nature, 580(7802), 239–244. https://doi.org/10.1038/s41586-020-2158-3
- Olguín, H. J., Guzmán, D. C., García, E. H., & Mejía, G. B. (2015). The role of dopamine and its dysfunction as a consequence of oxidative stress. Oxidative Medicine and Cellular Longevity, 2016(1). https://doi.org/10.1155/2016/9730467
- Ott, T., & Nieder, A. (2019). Dopamine and cognitive control in prefrontal cortex. Trends in Cognitive Sciences, 23(3), 213–234. https://doi.org/10.1016/j.tics.2018.12.006
- Raji, H., Dinesh, S., & Sharma, S. (2025). Inside the impulsive brain: a narrative review on the role of neurobiological, hormonal and genetic factors influencing impulsivity in psychiatric disorders. The Egyptian Journal of Neurology Psychiatry and Neurosurgery, 61(1). https://doi.org/10.1186/s41983-024-00930-9
- Schultz, W. (2016). Dopamine reward prediction error coding. Dialogues in Clinical Neuroscience, 18(1), 23–32. https://doi.org/10.31887/dcns.2016.18.1/wschultz
- Seo, D., Patrick, C. J., & Kennealy, P. J. (2008). Role of serotonin and dopamine system interactions in the neurobiology of impulsive aggression and its comorbidity with other clinical disorders. Aggression and Violent Behavior, 13(5), 383–395. https://doi.org/10.1016/j.avb.2008.06.003
- Stolyarova, A., O’Dell, S. J., Marshall, J. F., & Izquierdo, A. (2014). Positive and negative feedback learning and associated dopamine and serotonin transporter binding after methamphetamine. Behavioural Brain Research, 271, 195–202. https://doi.org/10.1016/j.bbr.2014.06.031
- Stott, S., & Ang, S. (2013). The generation of midbrain dopaminergic neurons. In Elsevier eBooks (pp. 435–453). https://doi.org/10.1016/b978-0-12-397265-1.00099-x
- Uhl, G. R., Koob, G. F., & Cable, J. (2019). The neurobiology of addiction. Annals of the New York Academy of Sciences, 1451(1), 5–28. https://doi.org/10.1111/nyas.13989